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Although thought to be inherited, I suspect it can also show up with too much exposure to glycol ether, and that it would be exposure to glycol ether in parent/s that would cause gene damage & such an autoimmune metabolic disorder in the first place.
Carnitine palmitoyltransferase II deficiency is a metabolic disorder characterized by an enzymatic defect that prevents long-chain fatty acids from being transported into the mitochondria for utilization as an energy source. The adult myopathic form of this disease was first characterized in 1973 by DiMauro and DiMauro. It is the most common inherited disorder of lipid metabolism affecting the skeletal muscle of adults. CPT II deficiency is also the most frequent cause of hereditary myoglobinuria. Symptoms of this disease are commonly provoked by prolonged exercise or periods without food. Can see symptoms of weakness / deep joint pain / chest pains. Fatty Acid Oxidation Disorders [PDF]
WHY Avoid add-on exposures to glycol ether chemicals *
Dr James D. Sidman: "Be a lumper not a splitter" (Look at the BIG PICTURE / not separate into its component parts)
Per definition, doctors do not know what the fatigue is of Chronic Fatigue Immune Dysfunction Syndrome (CFS, FM also) Find AIHA or IMHA, THE FATIGUE of CFIDS
Exposure to glycol ether looks like 'the flu'